In previous posts we suggested that the rare respiratory disease that is being called “Covid-19” may be caused by something else than a “new coronavirus” so many people are being tested positive for.
To be sure, recent news have been giving even more credence to this. To cite but one example, please consider this Reuters’ article (archive 1, archive 2). Titled “In four U.S. state prisons, nearly 3,300 inmates test positive for coronavirus — 96% without symptoms“, the article goes on to say officials are “very surprised” and “shocked” so many “positive” cases are “asymptomatic” (i.e. healthy).
What this means is that, out of a large “infected” population sample, 4% had flu-like symptoms. To grasp the absurdity of this, imagine an article titled “out of all people with brown hair, 4% had the flu”; you would think the two things perhaps aren’t correlated. Still, these articles are interesting to us, not only because they help falsify the virus narrative, but also because they confirm that we are expected to understand the threat was greatly exaggerated. In this case there is something else, namely the number “33”, which we will be covering in future posts about the esoteric and occult dimension of the coronacircus.
For the time being, the thread we have been unwinding is this one: a rare, peculiar and serious acute respiratory ailment has been happening in the world for at least a few years. In Brescia in 2018, it was labelled “legionella”. In the USA in 2019, it was called “vaping illness”. And across the world in 2020, it is being blamed on a “new coronavirus”. Whatever the principal cause, we have shown it seems correlated with atmospheric particulates (pollution).
Covid and Tuberculosis
A key to recognizing the disease across its various apparitions, apart from its specific clinical manifestations, is the very peculiar fact two thirds of those affected are men. It is true for the vaping illness; it is true for the Brescia pneumonia; it is true for “Covid-19”.
Tuberculosis is a very old disease; as old as civilization itself. Most cases are pulmonary, i.e. don’t spread outside of the lungs. It is thought to be caused by at least 9 different bacteria, which have been grouped in a single “complex” (Mycobacterium tuberculosis) because they are believed to all be able to cause the same disease.
Most infections by these bacteria produce no symptoms, a condition labelled latent tuberculosis, which is quite common as one quarter of the world population lives with it as of 2018. The vast majority of those people will never go to develop any symptoms.
The symptoms of “active” pulmonary tuberculosis are persistent coughing, loss of appetite, weight loss, fever and chills. Coughing up blood, a symptom popularly featured in movies, is actually rare.
Risk factors include smoking (increases the risk 2-4x) and diabetes (3x). Silicosis, i.e. fibrosis of the lungs caused by chronic inhalation of fine dust (silica particulates), increases the risk 30-fold; that lung disease is very often confused with pneumonia and tuberculosis itself.
Active pulmonary tuberculosis was also shown to be sometimes linked with hypoxemia, interstitial fibrosis and acute respiratory failure; the exact symptoms of critical “Covid” cases (archive 1, archive 2, full paper).
So, in other words, there are strange similarities between pulmonary tuberculosis and the rare disease lately being labelled “Covid”; quite striking is the the mysterious fact both ailments affect men in the same (dis)proportion. All this would remain anecdotal if there wasn’t a body of research showing tuberculosis is an important risk factor for Covid. Most interestingly, the turberculosis vaccine (BCG vaccination) is thought to reduce morbidity and mortality for Covid-19.
We share below some of this research, with excerpts (emphasis ours), and as always the full papers linked.
Tuberculosis and coronavirus: what do we know?
When comparing the state of MTB [Mycobacterium tuberculosis] infection among cases of pneumonia caused by COVID-19 with slight to moderate severity and with severe to critical severity, MTB coinfection was found to be lower in the first group (22%) in comparison to the second (78%), with statistical difference between them (p=0.005). These findings, although preliminary, point to the need to assess whether MTB infection is a risk factor COVID-19 and whether a causal relationship exists.
Covid and BCG Vaccination
Correlation between universal BCG vaccination policy and reduced morbidity and mortality for COVID-19: an epidemiological study
COVID-19 has spread to most countries in the world. Puzzlingly, the impact of the disease is different in different countries.
We found that countries without universal policies of BCG vaccination (Italy, Nederland, USA) have been more severely affected compared to countries with universal and long-standing BCG policies.
We also found that BCG vaccination also reduced the number of reported COVID-19 cases in a country. The combination of reduced morbidity and mortality makes BCG vaccination a potential new tool in the fight against COVID-19.
Tuberculosis Strongly Linked With Covid Severity
Active or latent tuberculosis increases susceptibility to COVID-19 and disease severity
Objective: To determine if latent or active TB increase susceptibility to SARS-COV-19 infection and disease severity, and lead to more rapid development of COVID-19 pneumonia.
Cases were grouped according to COVID-19 pneumonia severity (mild/moderate, severe/critical), and MTB infection status compared.
MTB co-infection linked with disease severity (severe/critical 78% vs mild/moderate cases 22%; p=0.0049), and rate of disease progression: infection to development of symptoms (MTB+SARS-CoV-2: 6.5+/-4.2 days vs SARS-COV-2: 8.9+/-5.2 days; p=0.073); from symptom development to diagnosed as severe (MTB+SARS-CoV-2: 3.4+/-2.0 days vs SARS-COV-2: 7.5+/-0.5 days; p=0.075).
WHO In Denial
If you read the letter carefully though, you notice it is merely addressing the research papers linking BCG vaccination policies and Covid prevalence (accusing them of ignoring confounder biases). The WHO is patently ignoring the research that establishes tuberculosis as the most significant cofactor.
As far as we are concerned, this is dishonest on the part of the WHO, and it is an attempt to muddy the waters. It will cause people to dismiss any link between the two conditions, while addressing only half of the argument.
Dr. Lawrence Broxmeyer MD
One doctor is going further, and postulating the mycobacteria tuberculosis is the actual origin of the infection, hospitalizations and deaths attributed to the “new coronavirus”. We have been made aware of his work by a reader, which we would like to wholeheartedly thank.
That doctor’s name is Lawrence Boxmeyer. Internist and medical researcher from Pennsylvania, he was on the staff at New York affiliates of Downstate, Cornell and NYU for 14 years. His publications are available on academia.edu, at this link. We shall link and quote some of his material here.
Further evidence tuberculosis bacterium masquerades as Covid-19 coronavirus.
According to Dr. Broxmeyer, COVID-19 may be nothing more than a passenger virus while the mycobacterium commonly known as tuberculosis, is wreaking all the damage in the lungs in the current epidemic that is holding the world in a news media-created grip of terror. Cause and effect have not been proven.
Mycobacteria tuberculosis is the great masquerader. Just type in “tuberculosis” and “masquerade” into your web browser and see how many maladies TB pretends to be. The TB mycobacterium has fooled doctor after doctor. And it may be fooling the entire world now.
Mycobacteria tuberculosis (TB) acts like a virus. It is a seasonal infection peaking in winter just like cold and flu viruses. While the TB mycobacterium is spread throughout the year, it is only when vitamin D levels are low and the immune system weak that it produces symptoms. It is a cell-wall deficient germ that appears like a virus under a microscope. It attacks the lungs, resulting in inflammation that essentially drowns infected patients who cannot breathe, just like COVID-19 coronavirus is said to do.
The COVID-19 coronavirus epidemic in China occurred while China had been battling a rampant TB outbreak for many months.
Inexplicably, there are far more cases of COVID-19 in Italy than other European countries. These cases of COVID-19 coronavirus infection occurred, like in China, in a background outburst of TB infections.
Dr. Broxmeyer further argues of a link between migration patterns, tuberculosis and Covid. Reading that whole interview is highly recommended.
Italy in Crisis
By 2013, Faccini et al reported in Emerging Infectious Diseases, an outbreak of tuberculosis, Beijing strain, in a primary school in Milan, Italy which was eventually traced to include 15 schoolchildren with active TB and 173 with latent infection.
Traditionally, Italy has a low incidence of tuberculosis (TB); and in 2008, the incidence of notified cases was only 7.6/100,000 population. Yet even by 2009, in Milan, the largest urban area and the birthplace of Italian COVID-19, in Lombardy, the incidence climbed steeply to 20.44/100,000 population. By 2019, Cuomo., et al. attributed this to rising immigration patterns.
Thus, just before the event attributed to COVID-19 in Northern Italy began, a deadly combination of rising TB rates followed by the introduction of porcine [from pigs] M. avium [also called fowl tuberculosis] into the environment would eventually first bring the Italian Northern provinces, and then the entire peninsula to its knees. This precise series of events led to the Great Pandemic of 1918 at Fort Funston and the Chinese episode at Wuhan, a major player in China’s pig industry.
Mycobacterium avium complex (MAC) or mycobacterium avium is a poorly understood disease which fulfills almost all of those characteristic signs and symptoms attributed to the latest “novel” Coronavirus.
When it appears in the lungs, Mycobacterium avium favors an older population with an underlying condition.
Non-tubercular-mycobacteria (NTM) such as Mycobacterium avium can be asymptomatic or can cause symptoms similar to tuberculosis, such as cough, fever, fatigue, and weight loss.
It is projected that the present Italian outbreak and outbreaks worldwide will follow the timetable of Yang’s Wuhan study, which describes an annual TB surge in Wuhan as being fueled by increased transmission in the winter; peaking in March, with a second smaller peak in September.
Again, this paper is fascinating, and we recommend reading it in full.
Promising Antimicrobial Hope for “Coronavirus”, but is it Working Against A Virus?
Azithromycin, an antibiotic with no antiviral activity whatsoever, is proving quite efficacious in treating the disease behind the “COVID-19” pandemic. Azithromycin is a first-line drug against Mycobacterium avium, which can simulate obstructive lung disease, and every other sign or symptom of COVID-19 documented to date. In addition azithromycin is a second or third-line drug against drug-resistant Mycobacterium tuberculosis.
In the United States, pulmonary Mycobacterium avium complex (MAC) disease or “fowl tuberculosis” is more common than tuberculosis. Furthermore, Mycobacterium avium complex is now the leading mycobacterial cause of chronic pneumonia in the United States.
Not only does the drug hydroxychloroquine inhibit intracellular TB, but it acts synergistically against mycobacterial disease when combined with certain antimycobacterials. Azithromycin is also used as an antimycobacterial.
This one is the most fascinating of Broxmeyer’s papers so far. For the sake of brevity, we have only quoted a small part above. Again we recommend reviewing it in full.
Here is one last article by Dr. Broxmeyer, no less fascinating that the others, that compares Covid-19, SARS, MERS and the Great Pandemic of 1918 with some surprising results and similarities.
Two things are happening, which we have been documenting in parallel.
First, the causality between a “new coronavirus” and the respiratory disease lately called “Covid” is being put into serious question. The fact so many people are infected (much more than previously thought) and so few people are affected (much less than previously thought) is a strong argument; the strongest however is that the disease seems to have manifested itself, as we documented, well before this latest “new coronavirus outbreak”.
Second, new causal factors are coming to light. We have previously made a strong case for atmospheric particulate pollution; today, we have raised the serious hypothesis of tuberculosis bacteria. As far as we can tell, we are the first to notice the fact that disease, just like Covid, mysteriously affects two-thirds of men.
Still, we are not yet fully satisfied. This “epidemic”, or at least the global response to it, seems to have indeed been planned long in advance. We are still looking for another factor, something newer, that could have served as a catalyst or a trigger in 2020.
Stay tuned, as there is more to come.